Stylised brain, ECG and mountain illustration
Consultant Neurosurgeon · Pre-Hospital Care Specialist

Professor Mark Wilson OBE

PhD · MBBChir · FRCS(Ed) · FIMC · MRCA · FRGS
Imperial College Healthcare NHS Trust · Imperial College London
Air Ambulance Kent, Surrey and Sussex

Neurotrauma Pre-Hospital Care Intracranial Pressure GoodSAM Co-Founder Kent Surrey & Sussex Air Ambulance SBNS Elected Council Member
Patient Information About & Research
>100
Peer-reviewed publications
1.4M
People on the GoodSAM platform in the UK
800K
NHS volunteers mobilised (COVID-19)
3×
Cardiac arrest survival uplift in clinical studies

Background

About Me

I am a Consultant Neurosurgeon at Imperial College Healthcare NHS Trust (St Mary's and Charing Cross Hospitals) and Professor of Practice (Neurosurgery) at Imperial College London, where I have established the Neurotrauma unit at St Mary's. I am also Co-Director of the Imperial Neurotrauma Centre.

In addition to my specialty interests in traumatic and hypoxic brain injury, I have clinical interests in all conditions related to intracranial pressure, including Normal Pressure Hydrocephalus (NPH), Idiopathic Intracranial Hypertension (IIH), and Hydrocephalus.

Alongside my surgical practice, I have been a practising pre-hospital care physician for 22 years — 18 years with London's Air Ambulance and 7 years with Kent Surrey & Sussex Air Ambulance, where I currently work 3 shifts per month. Pre-hospital neurosurgical emergencies — from roadside resuscitation to on-scene decision-making — sit at the heart of my clinical and research work.

My broader research interests span the full spectrum of neurotrauma and intracranial pressure physiology. I hold a PhD from UCL on the physiology of the brain at altitude and in hypoxia, and have been a researcher on expeditions to Everest, Cho Oyu, Bhutan, and the Arctic.

I co-founded GoodSAM (Good Smartphone Activated Medics), a globally deployed emergency response platform now used by 1.4 million people in the UK. What began as a tool to alert trained volunteers to cardiac arrests — tripling survival rates in clinical studies — has grown into a transformative platform across emergency services. GoodSAM now powers the NHS 111 video consultation system for England, enabling real-time triage and video-guided CPR. In the COVID-19 response, GoodSAM co-ordinated the mobilisation of 800,000 NHS volunteers — the largest single volunteer mobilisation in NHS history.

Beyond healthcare, GoodSAM is reshaping policing: working with police forces we are tripling domestic violence arrest rates by enabling rapid officer alerting and video evidence capture. The platform is also deployed by fire services in the US and internationally. GoodSAM is active across ambulance services, police, fire, mental health triage, and mass casualty co-ordination in the UK, Australia, New Zealand, USA, Canada, and beyond.

This page is intended simply to provide a little background about me and some information about the conditions I may be treating for my NHS patients. I do not do private work or medico-legal work — I simply don't have the time — so this is not a site for promoting services, and I do not appear on doctor comparison websites.

  • Consultant Neurosurgeon, Imperial College Healthcare NHS Trust
  • Professor of Practice (Neurosurgery), Imperial College London
  • Senior Pre-Hospital Care Specialist, Kent Surrey & Sussex Air Ambulance
  • Co-founder & Medical Director, GoodSAM
  • Elected Council Member, Society of British Neurological Surgeons
  • NHS Clinical Reference Group — Trauma & Burns (member)
  • NHS Clinical Reference Group — Neurosurgery (member)
  • ICRC Faculty & Neurosurgery Advisor

Recognition

OBE — For Services to Emergency Care & COVID-19 Response
2020
Principally awarded for the GoodSAM COVID-19 volunteer mobilisation programme.
Honorary Fellow, Queen Mary University London
2023
NHS Innovation of the Year
2017
Also: HSJ Award (2019).
Air Ambulance Doctor of the Year
AAA · 2017
Prime Minister's Point of Light Award
2014
Outstanding voluntary contribution to the community.

Guidance & Resources

Information for Patients

It is a privilege to undertake the work I do. Whether it is surgery for trauma, hydrocephalus, or raised intracranial pressure, the goal is almost always the same — to keep the person the same person they were before. Please always discuss your individual situation with your clinical team.

Traumatic Brain Injury

Traumatic brain injury (TBI) occurs when a sudden physical impact disrupts normal brain function. Injuries range from mild concussion — a temporary change in brain function — through to severe TBI involving structural damage, bleeding, or swelling within or around the brain. Our team has led the development of a new TBI classification system adopted by the Society of British Neurological Surgeons ↗.

Many patients with head injuries are found to have a chronic subdural haematoma — a collection of blood on the surface of the brain that develops gradually. If this is your diagnosis, useful patient information is available at chronicsubdural.info ↗.

Symptoms to watch for

  • Headache, particularly worsening headache
  • Nausea or vomiting
  • Confusion, disorientation or unusual behaviour
  • Unequal pupil size
  • Weakness or numbness in limbs
  • Difficulty speaking or understanding speech
  • Seizures
  • Loss of consciousness or decreasing level of consciousness

When to seek immediate help

  • Call 999 if the person becomes unconscious
  • Any seizure following a head injury
  • Worsening or very severe headache
  • Repeated vomiting
  • Clear fluid from nose or ears
  • Difficulty waking the person
  • High-risk injury mechanism (fall >1m, high-speed collision)

Post Head Injury Advice (NICE Guidelines)

The following advice is based on NICE clinical guideline CG176 (Head Injury). It is intended for patients discharged from hospital or Emergency Department following a head injury assessed as low risk.

You should rest and avoid strenuous activity for at least 24–48 hours. It is normal to experience mild headache, tiredness, difficulty concentrating, and slight dizziness in the days following a head injury — these symptoms usually resolve within two weeks.

You should NOT: drink alcohol, take sedative medications, drive a vehicle, operate machinery, or be left alone for the first 24 hours unless a responsible adult is with you.

If symptoms do not improve within two weeks, or if you develop new symptoms, please contact your GP or return to hospital.

Return immediately to hospital if: the person cannot be woken; has a seizure; develops weakness in arms or legs; becomes increasingly confused or behaves unusually; has a severe worsening headache unrelieved by paracetamol; vomits more than once; develops problems with vision, speech, hearing or balance.

Hydrocephalus

Hydrocephalus is a condition in which cerebrospinal fluid (CSF) — the fluid that surrounds and cushions the brain and spinal cord — accumulates abnormally within the ventricles (fluid-filled cavities) of the brain, causing them to enlarge. In adults, hydrocephalus most commonly arises as a result of obstruction to CSF flow, impaired absorption of CSF, or, rarely, overproduction. Common causes in adults include previous haemorrhage (bleeding into the brain or its coverings), meningitis, head injury, tumours, and aqueductal stenosis — a narrowing of the channel connecting the third and fourth ventricles. In many cases no cause is found.

The symptoms of hydrocephalus depend on how quickly pressure builds and the underlying cause, but commonly include headache (often worse in the morning or when lying down), nausea and vomiting, visual disturbance, unsteadiness, and cognitive slowing. Urgent treatment is required when pressure is high, as untreated hydrocephalus can cause serious and permanent neurological injury.

Investigations

  • MRI brain — the key investigation; defines ventricular size, identifies the site of obstruction, and excludes underlying causes such as tumour
  • CT brain — used when MRI is not possible, or in urgent situations
  • MRI CSF flow studies — to assess flow through the aqueduct and identify the level of obstruction
  • Lumbar puncture — to measure CSF pressure and, in selected cases, assess response to drainage
  • Further imaging — depending on the suspected underlying cause

Who is affected

  • Hydrocephalus can develop at any age in adulthood
  • Post-haemorrhagic hydrocephalus — following subarachnoid or intraventricular haemorrhage
  • Post-infectious — following bacterial meningitis or other CNS infections
  • Tumour-related — obstruction of CSF pathways by brain tumours
  • Aqueductal stenosis — may present in adulthood after years of compensation
  • Post-traumatic — following significant head injury
  • Idiopathic — no identifiable cause found

Treatment Option 1: VP Shunt (Ventriculoperitoneal Shunt)

A VP shunt is the most widely used surgical treatment for hydrocephalus. A small catheter is passed into the enlarged ventricle through a tiny hole in the skull, and connected via a pressure-regulated valve under the scalp to a second catheter that runs under the skin to the abdomen, where excess CSF drains safely into the peritoneal cavity and is reabsorbed. Surgery is performed under general anaesthetic and typically requires 2–3 days in hospital.

Benefits of VP shunting

  • Effective for almost all forms of hydrocephalus regardless of cause
  • Rapid reduction in intracranial pressure and symptom relief in most patients
  • Adjustable valves allow pressure settings to be changed non-invasively after surgery
  • Well-established procedure with decades of evidence

Risks of VP shunting

  • Shunt infection (3–5%) — often requiring shunt removal, antibiotic treatment, and later replacement
  • Shunt blockage — may occur at any time; requires revision surgery
  • Over-drainage — causing low-pressure headaches, or subdural haematoma (blood collection on brain surface)
  • Under-drainage — insufficient reduction of pressure
  • Haemorrhage at catheter insertion (rare: <1%)
  • Shunt dependency — once inserted, many patients rely on the shunt indefinitely
  • General anaesthetic risks

Treatment Option 2: Endoscopic Third Ventriculostomy (ETV)

ETV is a minimally invasive endoscopic procedure that can be an alternative to shunting in selected patients — particularly those with obstructive hydrocephalus (where CSF flow is blocked, rather than impaired absorption). A small camera (endoscope) is passed through a tiny hole in the skull into the third ventricle, and a small opening is made in the floor of the ventricle, creating a new pathway for CSF to bypass the obstruction and flow to where it can be reabsorbed naturally. No implant is left behind.

Benefits of ETV

  • Avoids the need for a permanent implanted shunt
  • No hardware to block, fracture, or become infected
  • If successful, provides a more physiological solution — restoring natural CSF circulation
  • Shorter operative time and recovery in many cases
  • Success rates of 70–90% in well-selected patients (aqueductal stenosis in particular)

Risks of ETV

  • Not suitable for all types of hydrocephalus — communicating or post-haemorrhagic hydrocephalus has lower ETV success rates
  • ETV failure — occurs in 10–30% of cases, most commonly in the first weeks to months; may require shunt insertion
  • Haemorrhage — risk of bleeding from basilar artery or other vessels near the floor of the third ventricle (rare but serious: <1%)
  • CSF leak or infection (uncommon)
  • Memory disturbance — due to proximity to fornix and hypothalamic structures (rare)
  • Late ETV closure — the stoma can close months or years later, causing recurrent hydrocephalus
The choice between VP shunt and ETV depends on the type and cause of hydrocephalus, your age, and other clinical factors. This will be discussed carefully with you before any decision is made. Both procedures aim for the same goal — reducing pressure and protecting brain function.

Normal Pressure Hydrocephalus

Normal Pressure Hydrocephalus (NPH) is a condition in which cerebrospinal fluid (CSF) — the fluid that surrounds and cushions the brain and spinal cord — accumulates in the ventricles (fluid-filled spaces) of the brain, causing them to enlarge. Despite this enlargement, pressure measurements of the CSF are often within the normal range, which gives the condition its name. NPH primarily affects older adults and is characterised by a triad of symptoms.

Classic symptom triad

  • Gait disturbance — a broad-based, shuffling, "magnetic" walk; difficulty lifting feet; falls
  • Urinary urgency and incontinence — difficulty controlling the bladder
  • Cognitive impairment — slowing of thought, forgetfulness, difficulty with complex tasks (sometimes misdiagnosed as dementia)

Investigations

  • MRI brain — to assess ventricular size and exclude other causes
  • CT brain — if MRI is not possible
  • Lumbar puncture (LP / Tap test) — CSF is drained to assess whether symptoms improve temporarily
  • Extended lumbar drainage — used in some centres to predict shunt response
  • CSF flow studies — in selected cases

The Tap Test (Lumbar Puncture)

The tap test is a key diagnostic procedure. A lumbar puncture is performed under local anaesthetic to remove approximately 30–50ml of CSF from around the lower spine. You will be assessed walking and performing cognitive tasks before and 24–48 hours after the procedure. A significant improvement in your walking suggests you are likely to respond well to a shunt operation.

After a lumbar puncture you may experience: headache (often worse when sitting or standing — this usually settles with lying down, rest, and oral fluids), back discomfort, and occasionally nausea. Rare complications include infection (<1%) or persistent headache requiring a blood patch.

Shunt Surgery — Treatment & Risks

If investigations suggest you are likely to respond to treatment, a ventriculoperitoneal (VP) shunt or lumboperitoneal (LP) shunt is inserted. This is a small tube that drains excess CSF from the brain to the abdominal cavity, where it is safely reabsorbed. Surgery is performed under general anaesthetic and typically requires a 2–3 day hospital stay.

Benefits

  • Improvement in walking in 60–80% of carefully selected patients
  • Improvement in bladder control in approximately 80%
  • Cognitive benefit — especially in earlier disease
  • Potential for significant improvement in quality of life

Risks of shunting

  • Shunt infection (2–5%) — may require shunt removal and replacement
  • Shunt blockage (10–15 years) — requiring revision surgery
  • Over-drainage — causing low-pressure headaches or subdural haematoma
  • Haemorrhage at shunt insertion (rare: <1%)
  • General anaesthetic risks
  • No improvement or deterioration (20–40%)

Idiopathic Intracranial Hypertension

Idiopathic Intracranial Hypertension (IIH) — also known as pseudotumour cerebri — is a condition in which the pressure of the CSF around the brain is persistently elevated without an obvious underlying cause such as a tumour or blockage. It predominantly affects women of childbearing age who are overweight, although it can occur in anyone. If untreated, IIH can cause permanent visual loss.

Symptoms

  • Persistent, severe headache — often worse in the morning or with lying down
  • Transient visual obscurations (brief greying or blackouts of vision)
  • Double vision (diplopia) — caused by pressure on the sixth cranial nerve
  • Pulsatile tinnitus — whooshing sound in the ears in time with the heartbeat
  • Visual field loss — if untreated, can become permanent
  • Neck and back pain

Investigations

  • MRI brain with MR venography — to exclude secondary causes and assess for venous sinus stenosis
  • Fundoscopy / OCT — to assess for papilloedema (optic disc swelling)
  • Formal visual field testing
  • Lumbar puncture — to measure opening pressure (elevated in IIH) and drain CSF for symptom relief
  • Blood tests — to exclude secondary causes

Treatment Options

Management of IIH is individualised. Many patients improve significantly with weight loss and medical treatment. Surgical options are reserved for those who do not respond, or who have severe or rapidly deteriorating vision.

Medical & conservative treatment

  • Weight loss — the most effective intervention where applicable; 5–10% body weight loss can lead to remission
  • Acetazolamide (Diamox) — reduces CSF production; first-line medication
  • Topiramate — alternative medication with weight loss benefit
  • Serial lumbar punctures — for acute symptom relief
  • Optic nerve sheath fenestration — to protect vision

Surgical options & risks

  • Ventriculoperitoneal (VP) or lumboperitoneal shunt — diverts CSF; risks similar to NPH shunting
  • Venous sinus stenting — for patients with significant venous sinus stenosis on MR venography; endovascular procedure
  • Risks: shunt infection, over-drainage, shunt revision, stent thrombosis (rare)
  • All surgical decisions are made in the context of an MDT (multidisciplinary team)
Important: IIH can cause permanent visual loss. If you notice sudden deterioration in your vision, contact your neurosurgical or neuro-ophthalmology team urgently or attend the Emergency Department.

Cranial Reconstruction (Cranioplasty)

A cranioplasty is an operation to replace a section of the skull that has been removed (a procedure called a decompressive craniectomy) or lost through trauma, infection, or disease. The skull defect may be repaired using the patient's own stored bone or — more commonly — with a custom-made synthetic implant (typically titanium mesh, PEEK polymer, or hydroxyapatite cement). The operation aims to protect the brain, restore the normal appearance of the head, and can also improve neurological function.

What the operation involves

  • Performed under general anaesthetic
  • The previous scalp scar is reopened and the skull defect is exposed
  • A custom-made implant or the stored bone flap is fixed in place with small titanium plates and screws
  • The scalp is carefully closed in layers
  • Hospital stay is typically 3–5 days
  • CT scan is usually performed before discharge

Timing

  • Usually performed 3–6 months after the initial craniectomy
  • Delayed to allow brain swelling to resolve and the patient to stabilise
  • Timing is individualised based on recovery and clinical condition
  • Earlier surgery may be considered in selected cases

Risks of Cranioplasty

Cranioplasty is generally well tolerated but, like any neurosurgical procedure, carries risks that will be discussed with you in detail during your consent process. The main risks include:

Specific risks

  • Infection — the most important complication; occurs in 5–15% of cases; may require implant removal and prolonged antibiotic treatment or replacement at a later stage
  • Brain injury / haemorrhage — risk of new bleeding around the brain during surgery, particularly if the brain was very adherent to the dura or skull
  • Seizures — risk of new or increased seizures in the post-operative period; anti-epileptic medication may be prescribed
  • Cosmetic deformity — the repaired contour may not be perfectly symmetrical; in some cases further adjustment surgery may be considered
  • Implant failure or resorption — stored bone may be resorbed (particularly in young patients or if the bone was infected), requiring a synthetic replacement
  • Further surgery — a proportion of patients (15–25%) will require at least one further operation due to infection, implant problems, or cosmetic concerns

General surgical risks

  • Wound healing problems or haematoma
  • Anaesthetic risks — discussed with the anaesthetist
  • Deep vein thrombosis (DVT) or pulmonary embolism — preventative measures are taken
  • Neurological deterioration — uncommon; most patients either improve or remain stable
Signs of implant infection to watch for: increasing redness, swelling or warmth of the scalp; wound breakdown; fever; increasing headache. Contact your neurosurgical team immediately if any of these develop.

Intracranial Pressure & the Role of Venous Outflow

For 200 years, the Monro-Kellie doctrine described the skull as a rigid closed box: brain tissue, blood, and cerebrospinal fluid (CSF) share a fixed space, and an increase in any one must be compensated by a reduction in the others — or pressure rises. This elegant concept has underpinned neurosurgery and neuro-critical care ever since.

However, this traditional model treats all three components equally, which misrepresents reality. CSF is produced very slowly — about 0.35 ml per minute — while blood flows into the brain at roughly 700 ml per minute. It is the dynamic relationship between arterial inflow and venous outflow that most powerfully drives moment-to-moment changes in intracranial pressure. I proposed a revision to this doctrine — Monro-Kellie 2.0 — which re-centres the venous outflow as the dominant, often-overlooked regulator of ICP. You can read the full paper here ↗.

The Unifying Thread: Venous Hypertension

A striking feature of Monro-Kellie 2.0 is that a single physiological mechanism — impaired venous outflow from the brain — appears to connect several seemingly unrelated clinical conditions and environments. When venous blood cannot drain adequately, pressure backs up inside the skull even when the brain itself is structurally normal.

At Altitude

  • Ascending rapidly to high altitude causes the body to respond to low oxygen by increasing cerebral blood flow
  • This increased inflow, if not matched by venous outflow, raises intracranial venous pressure and ICP
  • The result is the headache, nausea, and cognitive impairment of acute mountain sickness — and, in severe cases, high-altitude cerebral oedema (HACE)
  • Tight-fitting clothing, cervical collars, or neck positions that obstruct jugular venous drainage can worsen this markedly
  • My research expeditions to Everest, Cho Oyu, Bhutan, and the Arctic explored these mechanisms directly

In Space (Microgravity)

  • In microgravity, the normal postural effect of gravity on venous drainage is lost — blood no longer preferentially drains away from the head
  • Venous pressure in the head rises, and ICP increases chronically — even in healthy astronauts
  • This is now recognised as a significant problem for long-duration spaceflight, causing visual changes, papilloedema, and a syndrome closely resembling IIH
  • The parallels between spaceflight-associated neuro-ocular syndrome and terrestrial conditions of venous outflow obstruction are a powerful validation of the Monro-Kellie 2.0 framework

Idiopathic Intracranial Hypertension (IIH)

  • IIH — raised ICP without an obvious structural cause — has long been puzzling
  • Monro-Kellie 2.0 offers a unifying explanation: many patients with IIH have stenosis (narrowing) of the venous sinuses inside the skull, limiting venous outflow
  • This creates a self-reinforcing loop — raised ICP compresses the sinus further, worsening drainage and raising pressure still more
  • Understanding this venous mechanism has opened the door to stenting of venous sinuses as a treatment — directly addressing the outflow obstruction rather than simply managing the pressure
  • Body weight and posture also influence venous drainage, explaining why IIH is closely associated with obesity

Traumatic Brain Injury (TBI)

  • After a head injury, the brain swells and bleeds — but venous outflow obstruction frequently compounds the injury
  • Cervical collar application, poor patient positioning, and airway management decisions can all significantly affect jugular venous pressure and therefore ICP
  • In the pre-hospital setting, these seemingly minor decisions can make a major difference to outcome — and have informed my clinical practice as an air ambulance doctor for 22 years
  • Refractory intracranial hypertension after TBI is often driven by a failure of venous outflow to match arterial inflow — not simply by brain swelling alone
  • Targeted management of venous drainage (head positioning, avoiding obstruction, sedation to reduce venous congestion) is now a core element of neurotrauma care

Why This Matters for Patients

The practical implication of Monro-Kellie 2.0 is that many patients across conditions as varied as IIH, altitude sickness, post-TBI, and spaceflight share a common physiological problem — impaired venous drainage — that is potentially modifiable. Recognising this link guides more precise treatment: whether that is stenting a venous sinus in IIH, carefully positioning a trauma patient on the roadside, or designing protective countermeasures for astronauts.

Read: Monro-Kellie 2.0 (Wilson MH, JCBFM 2016) — Full Paper

Emergency Response Platform

GoodSAM

Good Smartphone Activated Medics — a globally deployed emergency response platform I co-founded to transform pre-hospital care.

1.4M
People on the platform in the UK
800K
NHS volunteers mobilised during COVID-19
117+
Lives saved in NSW alone
3×
Cardiac arrest survival uplift in clinical studies

GoodSAM bridges the gap between the emergency call and the arrival of an ambulance — alerting the nearest trained volunteer to attend, often reaching the patient in seconds. With video-guided CPR, a call handler or GoodSAM responder can direct a bystander through resuscitation in real time, dramatically improving outcomes. GoodSAM is the NHS 111 video system for England — bringing clinical triage into the home.

In policing, GoodSAM is transforming how forces respond to domestic violence, missing persons, and major incidents — enabling rapid alerting, live video evidence, and co-ordinated deployment. Working with police services we are tripling domestic violence arrest rates. The platform is also deployed by fire services across the US and internationally for rapid crew alerting and incident co-ordination.

For full clinical evidence, case studies, and partner information, visit www.goodsamapp.org — or the Australian & New Zealand evidence and case studies at goodsamapp.org/oz.

GoodSAM in action

Cardiac Arrest Response
GoodSAM alerting trained volunteers to cardiac arrests — saving lives before the ambulance arrives.
Video-Assisted CPR
Real-time video guidance directing bystanders through CPR — the NHS 111 video system for England.
NSW Police Launch
GoodSAM deployed across NSW Police — transforming response to domestic violence and major incidents.
Crimewatch Feature
BBC Crimewatch feature on GoodSAM's role in policing — missing persons and live incident response.
Oklahoma Fire Service Launch
GoodSAM deployed by Oklahoma Fire — rapid crew alerting and incident co-ordination across the US.
ITN News — GoodSAM in Dorset
ITN national news feature on the life-saving benefits of GoodSAM, filmed with Dorset ambulance service.

News stories & evidence

NSW Government · April 2026 100 lives saved — NSW Ambulance GoodSAM milestone ↗ Bay Post · May 2026 GoodSAM volunteers bridge vital gap in NSW cardiac arrests — survivor story ↗ NSW Health · October 2025 80 lives saved, 11,600 volunteers in NSW — AED map launch ↗ NSW Ambulance Official survivor stories: Strangers Unite to Save Traveller's Life ↗ Tasmanian Government Ambulance Tasmania & GoodSAM — State Government Partnership ↗ GoodSAM.org Peer-reviewed evidence base — full publication list ↗

Lectures & Media

Talks

Selected public lectures, conference presentations, and media appearances covering neurotrauma, pre-hospital care, and emergency medicine innovation.

This is something of an old public talk (from when I was young!) but the sentiment counts today as much as ever.

Neurotrauma & Pre-Hospital Brain Injury Management
A lecture covering the science and clinical management of traumatic brain injury, with insights from pre-hospital and in-hospital care, and novel classification systems.
Teaching Children about Pre-Hospital Care & Neurosurgery
An educational session bringing the world of emergency medicine and neurosurgery to life for young people — explaining what happens when someone is seriously injured and how doctors help at the scene and in hospital.

Academic Output

Research & Publications

Over 100 peer-reviewed publications. First and last author work in NEJM, The Lancet, Lancet Neurology, Annals of Neurology, and JCBFM.

I am Principal Investigator on a number of collaborative clinical trials, and Chief Investigator on the NIHR-funded Spinal Immobilisation Study — www.spine.study — examining the evidence base for cervical spine immobilisation following blunt trauma.

Book

The Medic's Guide to Work and Electives Around the World — book cover
The Medic's Guide to Work and Electives Around the World ↗
Wilson MH (Author) · Arnold / Hodder · 3rd Edition
A motivational and practical guide to making the most of your time abroad — whether as a medical student planning an elective or a doctor seeking a longer-term change of scene. Covering over 100 countries, career suggestions from NASA research to the Flying Doctors, and practical advice on funding, health, and logistics. "Medicine is your passport to the world."

Get in Touch

Contact

For clinical referrals, please contact Imperial College Healthcare NHS Trust via the standard referral pathway. For speaking enquiries, academic collaborations, or GoodSAM partnership enquiries, please use the links below.

Imperial Neurotrauma Centre
imperialneurotrauma.co.uk
Academic Profile
Imperial College Profile ↗
GoodSAM Evidence Base
goodsamapp.org/evidence